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Advances in Dermatology and Allergology/Postępy Dermatologii i Alergologii
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Original paper

Apoptosis and PTX3 in lichen planus

Ibrahim H. Yavuz
1
,
Göknur Özaydın Yavuz
2
,
Nazlı Caf
2
,
Mustafa Tümtürk
2
,
Harbiye Dilek Canat
2
,
Mehmet Onur Gökalp
1

  1. Department of Dermatology, Cam and Sakura City Hospital, Istanbul, Turkey
  2. Department of Dermatology, Health Sciences University, Istanbul, Turkey
Adv Dermatol Allergol
DOI: https://doi.org/10.5114/ada.2024.145205
Online publish date: 2024/11/21
Article file
- Apoptosis.pdf  [0.13 MB]
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1. Boch K, Langan EA, Kridin K, et al. Lichen planus. Front Med (Lausanne) 2021; 8: 737813.
2. Sugerman PB, Savage NW, Walsh LJ, et al. The pathogenesis of oral lichen planus. Crit Rev Oral Biol Med 2002; 13: 350-65.
3. Zhang H, Wang R, Wang Z, et al. Molecular insight into pentraxin-3: update advances in innate immunity, inflammation, tissue remodeling, diseases, and drug role. Biomed Pharmacother 2022; 156: 113783.
4. Nauta AJ, Daha MR, van Kooten C, et al. Recognition and clearance of apoptotic cells: a role for complement and pentraxins. Trends Immunol 2003; 24: 148-54.
5. Mantovani A, Garlanda C, Doni A, et al. Pentraxins in innate immunity: from C-reactive protein to the long pentraxin PTX3. J Clin Immunol 2008; 28: 1-13.
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7. Peri G, Introna M. Corradi D, et al. PTX3, A prototypical long pentraxin, is an early indicator of acute myocardial infarction in humans. Circulation 2000; 102: 636-41.
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9. Souza DG, Amaral FA, Fagundes CT, et al. The long pentraxin PTX3 is crucial for tissue inflammation after intestinal ischemia and reperfusion in mice. Am J Pathol 2009; 174: 1309-18.
10. Augusto JF, Poli C, Beauvillain C, et al. Anti-pentraxin antibodies in autoimmune systemic diseases: focus on anti-pentraxin-3 autoantibodies. Int Rev Immunol 2017; 36: 145-53.
11. Ortega-Hernandez OD, Bassi N, Shoenfeld Y, et al. The long pentraxin 3 and its role in autoimmunity. Semin Arthritis Rheum 2009; 39: 38-54.
12. Wang L, Cano M, Datta S, et al. Pentraxin 3 recruits complement factor H to protect against oxidative stress-induced complement and inflammasome overactivation. J Pathol 2016; 240: 495-506.
13. Bevelacqua V, Libra M, Mazzarino MC, et al. Long pentraxin 3: a marker of inflammation in untreated psoriatic patients. Int J Mol Med 2006; 18: 415-23.
14. Rovere P, Peri G, Fazzini F, et al. The long pentraxin PTX3 binds to apoptotic cells and regulates their clearance by antigen-presenting dendritic cells. Blood 2000; 96: 4300-6.
15. Shiraki A, Kotooka N, Komoda H, et al. Pentraxin-3 regulates the inflammatory activity of macrophages. Biochem Biophys Rep 2016; 5: 290-5.
16. Inforzato A, Peri G, Doni A, et al. Structure and function of the long pentraxin PTX3 glycosidic moiety: fine-tuning of the interaction with C1q and complement activation. Biochemistry 2006; 45: 11540-51.
17. Shiva A, Arab S, Mousavi SJ, et al. Serum and salivary level of nitric oxide (NOx) and CRP in oral lichen planus (OLP) patients. J Dent (Shiraz) 2020; 21: 6-11.
18. Hashemy SI, Gharaei S, Vasigh S, et al. Oxidative stress factors and C-reactive protein in patients with oral lichen planus before and 2 weeks after treatment. J Oral Pathol Med 2016; 45: 35-40.
19. Sağ S, Kamanlı A, Tekeoğlu I, et al. The relationship of pentraxin-3 levels in Behcet disease with IL-17, disease activity and atherosclerotic risk factors. Ann Rheum Dis 2016; 75: 1087-8.
20. Kasperska-Zajac A, Grzanka A, Misiolek M, et al. Pentraxin-3 as a local inflammatory marker in chronic spontaneous urticaria. Cytokine 2015; 76: 566-8.
21. Halil Yavuz I, Ozaydın Yavuz G. The role of pentraxin 3 in pemphigus vulgaris. Adv Dermatol Alergol 2020; 37: 503-7
Copyright: © 2024 Termedia Sp. z o. o. This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-ShareAlike 4.0 International (CC BY-NC-SA 4.0) License (http://creativecommons.org/licenses/by-nc-sa/4.0/), allowing third parties to copy and redistribute the material in any medium or format and to remix, transform, and build upon the material, provided the original work is properly cited and states its license.
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